I've been involved in a lot of trials that have involved brain scanning. (example)
But
I've never been a big fan of using brain scans to try to predict the
deficit. In school we were taught to look at the artery that was blocked. The artery that was blocked leading to the brain or in the
brain was supposed to give you some indication about what deficits were.
I would suggest this is where neurology and physiatry on one hand-- and neuroscience on the other-- kind of disagree. The neurologist and phsyiatrist say that this info (brain scan and/or artery blocked) helps determine the deficit, and even the chance of recovery.
But neuroscientists would suggest that, because the brain wiring is "random access" (that any part of the brain may be wired to any other part of the brain), looking at a brain scan maybe grossly predictive, but really tells you not much at all.
In all my seminars I suggest that the thing to do is to examine the patient in a very limited way.
Here's suggestions that will give you global insight into the potential for recovery:
1. Can they move their hand? First, let's define "hand movement." I would suggest that hand movement is any movement from the wrist to fingers, in any direction. The easiest movement for most survivors is closing (fist) the hand. The problem is that many strokes survivors think that they can't close their hand. If they think they can't, do this:
I would suggest this is where neurology and physiatry on one hand-- and neuroscience on the other-- kind of disagree. The neurologist and phsyiatrist say that this info (brain scan and/or artery blocked) helps determine the deficit, and even the chance of recovery.
But neuroscientists would suggest that, because the brain wiring is "random access" (that any part of the brain may be wired to any other part of the brain), looking at a brain scan maybe grossly predictive, but really tells you not much at all.
In all my seminars I suggest that the thing to do is to examine the patient in a very limited way.
Here's suggestions that will give you global insight into the potential for recovery:
1. Can they move their hand? First, let's define "hand movement." I would suggest that hand movement is any movement from the wrist to fingers, in any direction. The easiest movement for most survivors is closing (fist) the hand. The problem is that many strokes survivors think that they can't close their hand. If they think they can't, do this:
- Bend (technically: Flex) the wrist. This will allow you to open the fingers.
- Put the elbow in more than 90° of extension (more straight than a "shake hands" position)
- With your fingers inside the palm ask the survivor to try to touch their chin.
- This will force the flexor synergy into play, and if they are able, the fingers will flex and you'll feel the pressure on your fingers. Voilà: they have hand movement! From that you can determine potential for recovery of the hand. (Hint: there is potential).
2. Brush both forearms
lightly with your fingertips. Ask the survivor if they feel the same,
and if not, how they feel different. If they do feel different, this
suggests that sensation has been hit. Try to get them to give you a
percentage ("My bad arm feels about 25% of my my good arm feels"). There
is a general association between tactile (brushing your fingertips) and
proprioception (feeling where that limb is in space). And
proprioception has huge implications for recovery.
3. Vision: Can they follow the tip of a pen with their head held straight in front of them all the way towards their affected side? If they tend not to follow things on the affected side, this suggests a hemi field cut and possibly "unilateral neglect" – an inability to attend to the affected side.
4. Speech: can they speak; do they understand what's being said?
5. Are they walking, how well, how fast are they walking?
There-- now you have global insight into that stroke survivor. Isn't that more important than knowing that an occlusion of the inferolateral arterial group infarcted the posterolateral thalamus?
3. Vision: Can they follow the tip of a pen with their head held straight in front of them all the way towards their affected side? If they tend not to follow things on the affected side, this suggests a hemi field cut and possibly "unilateral neglect" – an inability to attend to the affected side.
4. Speech: can they speak; do they understand what's being said?
5. Are they walking, how well, how fast are they walking?
There-- now you have global insight into that stroke survivor. Isn't that more important than knowing that an occlusion of the inferolateral arterial group infarcted the posterolateral thalamus?
3 comments:
This is where I'll have to disagree with you Peter, yes the connections make it almost impossible to tell what deficits you might have. I've never seen anything that tells the medical staff what the damage is in the white matter. But then I'm a computer programmer, I look for direct cause and effect.
Dean! I'm counting on you to disagree! Just keep me honest... So, there is cause and effect; brain damage leads to deficits. We agree that the scan will not tell you much about the deficits (although there is some general global stuff you can get). I think its a technological problem. You'd need "pre" data (prior to injury); a very detailed brain baseline, if you will. Then, once there is injury it would be compared to the pre-injured data and have a real sense of potential deficit. Maybe?
I like your idea. The reason I focus on actual damage is that the same deficit could be caused by at least 9 different scenarios and I would think that the proposed solution might be different.
Effect; Can't extend arm straight.
Possible causes;
1. motor cortex area controlling that is dead.
2. motor cortex area controlling that is damaged.
3. pre-motor cortex area controlling that is dead.
4. pre-motor cortex area controlling that is damaged.
5. executive control area controlling that is dead.
6. executive control area controlling that is damaged.
7. white matter underlying motor is dead.
8. white matter underlying motor is damaged.
9. Spasticity of the bicep is preventing extension.
I think too much and have been for the past 9 years.
Post a Comment